Latent Sinoatrial Conduction Disturbances in Symptomatic Patients with Wolff‐Parkinson‐White Syndrome
Identifieur interne : 000409 ( Main/Corpus ); précédent : 000408; suivant : 000410Latent Sinoatrial Conduction Disturbances in Symptomatic Patients with Wolff‐Parkinson‐White Syndrome
Auteurs : Peter Rakovec ; Primo Rode ; Japec Jakopin ; Matija HorvatSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1983-01.
English descriptors
Abstract
Sinoatrial conduction abnormalities were studied in fourteen patients with Wolff‐Parkinson‐White (WPW) syndrome and frequent attacks of supraventricular paroxysmal tachycardia. Sinoatrial conduction time (SACT), obtained by the method of constant atrial pacing, was prolonged in all but two patients. The mean value was 277 ± 56 ms (mean ± SD). SACT was measured in the same way in sixteen patients without signs of pre‐excitation or Sinoatrial disease; SACT was significantly shorter (p < 0.001) than in the WPW group of patients. The prolongation of SACT in patients with WPW syndrome was confirmed by measuring SACT by the alternative method of programmed atrial stimulation. SACT was found to be prolonged in eight patients (mean ± SD = 239 ± 47 ms). Whether our results should be attributed to WPW syndrome or frequent supraventricular tachycardias—both being a characteristic feature of our patients—remains uncertain. The results of this study, however, indicate that in symptomatic patients with WPW syndrome and frequent episodes of supraventricular tachycardia, Sinoatrial conduction is frequently impaired. Slackening of the sinoatrial conduction is a possible predisposing factor for initiation of tachycardias in the WPW syndrome.
Url:
DOI: 10.1111/j.1540-8159.1983.tb06573.x
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Sinoatrial conduction time (SACT), obtained by the method of constant atrial pacing, was prolonged in all but two patients. The mean value was 277 ± 56 ms (mean ± SD). SACT was measured in the same way in sixteen patients without signs of pre‐excitation or Sinoatrial disease; SACT was significantly shorter (p < 0.001) than in the WPW group of patients. The prolongation of SACT in patients with WPW syndrome was confirmed by measuring SACT by the alternative method of programmed atrial stimulation. SACT was found to be prolonged in eight patients (mean ± SD = 239 ± 47 ms). Whether our results should be attributed to WPW syndrome or frequent supraventricular tachycardias—both being a characteristic feature of our patients—remains uncertain. The results of this study, however, indicate that in symptomatic patients with WPW syndrome and frequent episodes of supraventricular tachycardia, Sinoatrial conduction is frequently impaired. Slackening of the sinoatrial conduction is a possible predisposing factor for initiation of tachycardias in the WPW syndrome.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>PETER RAKOVEC</name><affiliations><json:string>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</json:string></affiliations></json:item><json:item><name>PRIMOẐ RODE</name><affiliations><json:string>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</json:string></affiliations></json:item><json:item><name>JAPEC JAKOPIN</name><affiliations><json:string>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</json:string></affiliations></json:item><json:item><name>MATIJA HORVAT</name><affiliations><json:string>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Sinoatrial conduction</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>supraventricular tachycardia</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>sick sinus syndrome</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>WPW syndrome</value></json:item></subject><articleId><json:string>PACE2</json:string></articleId><language><json:string>eng</json:string></language><abstract>Sinoatrial conduction abnormalities were studied in fourteen patients with Wolff‐Parkinson‐White (WPW) syndrome and frequent attacks of supraventricular paroxysmal tachycardia. 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Sinoatrial conduction time (SACT), obtained by the method of constant atrial pacing, was prolonged in all but two patients. The mean value was 277 ± 56 ms (mean ± SD). SACT was measured in the same way in sixteen patients without signs of pre‐excitation or Sinoatrial disease; SACT was significantly shorter (p < 0.001) than in the WPW group of patients. The prolongation of SACT in patients with WPW syndrome was confirmed by measuring SACT by the alternative method of programmed atrial stimulation. SACT was found to be prolonged in eight patients (mean ± SD = 239 ± 47 ms). Whether our results should be attributed to WPW syndrome or frequent supraventricular tachycardias—both being a characteristic feature of our patients—remains uncertain. The results of this study, however, indicate that in symptomatic patients with WPW syndrome and frequent episodes of supraventricular tachycardia, Sinoatrial conduction is frequently impaired. Slackening of the sinoatrial conduction is a possible predisposing factor for initiation of tachycardias in the WPW syndrome.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>Sinoatrial conduction</term></item><item><term>supraventricular tachycardia</term></item><item><term>sick sinus syndrome</term></item><item><term>WPW syndrome</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1983-01">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/3076ECAA77CE9B18812DD6A5571F936603E15798/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="01001"><doi origin="wiley">10.1111/pace.1983.6.issue-1</doi><numberingGroup><numbering type="journalVolume" number="6">6</numbering><numbering type="journalIssue" number="1">1</numbering></numberingGroup><coverDate startDate="1983-01">January 1983</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0000200" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1983.tb06573.x</doi><idGroup><id type="unit" value="PACE2"></id></idGroup><countGroup><count type="pageTotal" number="6"></count></countGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="2">2</numbering><numbering type="pageLast" number="7">7</numbering></numberingGroup><correspondenceTo>Address fpr reprints: Peter Rakovec, M.D., Ph.D., Topnis̆ka 43, 61000 Ljubljana, Yugoslavia.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE2.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received October 21, 1981; revision received December 23, 1981; accepted January 8, 1982</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="24"></count><count type="linksCrossRef" number="5"></count></countGroup><titleGroup><title type="main">Latent Sinoatrial Conduction Disturbances in Symptomatic Patients with Wolff‐Parkinson‐White Syndrome</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>PETER</givenNames><familyName>RAKOVEC</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>PRIMOẐ</givenNames><familyName>RODE</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>JAPEC</givenNames><familyName>JAKOPIN</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>MATIJA</givenNames><familyName>HORVAT</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="SI"><unparsedAffiliation>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">Sinoatrial conduction</keyword><keyword xml:id="k2">supraventricular tachycardia</keyword><keyword xml:id="k3">sick sinus syndrome</keyword><keyword xml:id="k4">WPW syndrome</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Sinoatrial conduction abnormalities were studied in fourteen patients with Wolff‐Parkinson‐White (WPW) syndrome and frequent attacks of supraventricular paroxysmal tachycardia. Sinoatrial conduction time (SACT), obtained by the method of constant atrial pacing, was prolonged in all but two patients. The mean value was 277 ± 56 ms (mean ± SD). SACT was measured in the same way in sixteen patients without signs of pre‐excitation or Sinoatrial disease; SACT was significantly shorter (p < 0.001) than in the WPW group of patients. The prolongation of SACT in patients with WPW syndrome was confirmed by measuring SACT by the alternative method of programmed atrial stimulation. SACT was found to be prolonged in eight patients (mean ± SD = 239 ± 47 ms). Whether our results should be attributed to WPW syndrome or frequent supraventricular tachycardias—both being a characteristic feature of our patients—remains uncertain. The results of this study, however, indicate that in symptomatic patients with WPW syndrome and frequent episodes of supraventricular tachycardia, Sinoatrial conduction is frequently impaired. Slackening of the sinoatrial conduction is a possible predisposing factor for initiation of tachycardias in the WPW syndrome.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Latent Sinoatrial Conduction Disturbances in Symptomatic Patients with Wolff‐Parkinson‐White Syndrome</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Latent Sinoatrial Conduction Disturbances in Symptomatic Patients with Wolff‐Parkinson‐White Syndrome</title></titleInfo><name type="personal"><namePart type="given">PETER</namePart><namePart type="family">RAKOVEC</namePart><affiliation>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</affiliation><description>Correspondence: Address fpr reprints: Peter Rakovec, M.D., Ph.D., Topnis̆ka 43, 61000 Ljubljana, Yugoslavia.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">PRIMOẐ</namePart><namePart type="family">RODE</namePart><affiliation>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">JAPEC</namePart><namePart type="family">JAKOPIN</namePart><affiliation>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">MATIJA</namePart><namePart type="family">HORVAT</namePart><affiliation>Cardiological Clinic and Center of Intensive Internal Medicine, Clinical Center, and Institute of Physiology, Medical Faculty, Ljubljana, Slovenia, Yugoslavia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1983-01</dateIssued><edition>Received October 21, 1981; revision received December 23, 1981; accepted January 8, 1982</edition><copyrightDate encoding="w3cdtf">1983</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">24</extent></physicalDescription><abstract lang="en">Sinoatrial conduction abnormalities were studied in fourteen patients with Wolff‐Parkinson‐White (WPW) syndrome and frequent attacks of supraventricular paroxysmal tachycardia. Sinoatrial conduction time (SACT), obtained by the method of constant atrial pacing, was prolonged in all but two patients. The mean value was 277 ± 56 ms (mean ± SD). SACT was measured in the same way in sixteen patients without signs of pre‐excitation or Sinoatrial disease; SACT was significantly shorter (p < 0.001) than in the WPW group of patients. The prolongation of SACT in patients with WPW syndrome was confirmed by measuring SACT by the alternative method of programmed atrial stimulation. SACT was found to be prolonged in eight patients (mean ± SD = 239 ± 47 ms). Whether our results should be attributed to WPW syndrome or frequent supraventricular tachycardias—both being a characteristic feature of our patients—remains uncertain. The results of this study, however, indicate that in symptomatic patients with WPW syndrome and frequent episodes of supraventricular tachycardia, Sinoatrial conduction is frequently impaired. Slackening of the sinoatrial conduction is a possible predisposing factor for initiation of tachycardias in the WPW syndrome.</abstract><subject lang="en"><genre>Keywords</genre><topic>Sinoatrial conduction</topic><topic>supraventricular tachycardia</topic><topic>sick sinus syndrome</topic><topic>WPW syndrome</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1983</date><detail type="volume"><caption>vol.</caption><number>6</number></detail><detail type="issue"><caption>no.</caption><number>1</number></detail><extent unit="pages"><start>2</start><end>7</end><total>6</total></extent></part></relatedItem><identifier type="istex">3076ECAA77CE9B18812DD6A5571F936603E15798</identifier><identifier type="DOI">10.1111/j.1540-8159.1983.tb06573.x</identifier><identifier type="ArticleID">PACE2</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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